| Re:Kurz: Lern Einkaufen, Kochen, Essen. Lang: siehe Text |  |
>Ich bin kein Arzt und hab mir das alles zusammen gekratzt aus Dokus und Reportagen, immer mit der Frage im Hinterkopf ist das seriös. Das mit dem Zucker hab ich nicht mehr gefunden (mal schauen ob ichs noch finde), aber hier ist der Link zur Info von Salz.
>
>[https://youtu.be/1fSIRWD7FLM?t=189]
Ich habe mir mir mal das Originalpaper zu der im Video angegebenen Studie angeschaut.
[https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6070150/]
In dieser Pilotstudie wurde u.a eine bestimmte Lactobazillus-Untergruppe untersucht, die nicht in allen Menschen vorkommt (50%). Leider wird aus den Konkunktiven von den Medien schnell ein (vereinfachender) Indikativ gemacht. Erst später wird im obigen Video das auch bei anderen Studien/Sachverhalten hervorgehoben. Aber "zuviel Salz -> Multiple Sklerose" ist schon 'ne steile These.
Aus dem Original:
It is therefore currently unclear
whether high salt effects can be generalized beyond the actively induced
model and to multiple sclerosis. These limitations may also extend to
the recent controversy regarding the more general effects of a HSD
on neuroinflammation
These results highlight the microbiome as a
saltsensitive compartment but do not speak against a more direct
effect of high salt on host cells. Earlier investigations have shown that
the ionic microenvironment directly affects various immune cells38–41.
Salt intake also has profound actions on hormonal systems, such as the
renin–angiotensin–aldosterone axis.
Our pilot study in humans is limited in power and needs to be
validated in a larger cohort. Considering this limitation, it suggests that
even a moderate salt challenge may affect the persistence of intestinal
Lactobacillus strains and other bacteria, along with an increase in
proinflammatory T H 17 cells and saltsensitive blood pressure
changes. Interestingly, newborn infants have the greatest abundance
of Lactobacillus species, which decreases over time42,43
. Compared to microbiomes from indigenous populations, abundance of Lactobacillus
strains in ‘Western’ gut microbiomes is low44
. Salt ingestion that already starts at a young age may partially have contributed to the relative loss of Lactobacillus strains from Western microbiomes and thereby may have role in the development of hypertension and autoimmunity.
Finally, the development of microbiotatargeted therapies is an
intriguing new avenue for many diseases. Nevertheless, changes in
microbiome composition or function must first be carefully shown
to contribute to any disease. Our experimental data in mice suggest
that the gut microbiota might serve as a potential target to counteract
salt sensitive conditions. The identification of Lactobacillus as a ‘natural
inhibitor’ of high saltinduced T H17 cells in mice could serve as a basis
for the development of novel prevention and treatment strategies. It is
up to randomized controlled trials in humans with diseases to test this
hypothesis. Moreover, any future dietary salt intervention trial should
thus consider monitoring the microbiome to expand on our observations.
Das Salz auf das RAAS Einfluss hat ist nicht verwunderlich:
[https://de.wikipedia.org/wiki/Renin-Angiotensin-Aldosteron-System]